How pathological alpha synuclein causes neuron death
A new finding that pathological alpha-synuclein causes cells to increase protein synthesis suggests new targets for treating PD.
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A new finding that pathological alpha-synuclein causes cells to increase protein synthesis suggests new targets for treating PD.
New software can make protein molecules that bind with high affinity and specificity to many biomarkers, including human hormones.
A discovery about Zika’s enzyme, NS2B-NS3, offers promise for therapeutic targets for Zika and other flaviviruses.
In this eBook, discover how to better understand tissue landscapes with different microscopy techniques including multiplex imaging.
Using saturation genome editing, researchers have created a map of disease-causing mutations for neurodevelopmental disorders and cancer.
A discovery about the mutated protein DECTIN-1, that limits the production of T regulatory cells, could lead to more effective drugs.
New findings about how RBM10 inhibits lung cancer growth offers potential for an anti-cancer drug and more personalised treatment.
A signalling mechanism for ventricular fibrillation involving the stress kinases p38γ and p38δ has been discovered.
Findings of rare CRISPR-linked gene modules and a novel CRISPR-Cas system have promising implications for genomic therapeutics.
Quantum-Si's Next-Generation Protein Sequencing technology on Platinum™, rapidly distinguishes SARS-CoV-2 variants based on amino acid sequence.
Researchers have developed a new method to study protein clumps that occur in many difficult-to-treat diseases.
Researchers often encounter common problems during protein sample preparation which can compromise the quality and quantity of protein obtained. Explore our solutions to these common challenges, including sample handling, storage and preparation method issues.
New research identifies how trabeculae transform into ventricles, which could positively impact heart disease treatments.
Understanding how a molecular transport system inside neurons works could be used for therapeutic approaches to neurodegenerative disease.
New pathway involved in cellular protein degradation could develop LYTACs therapy and find new treatments for lysosome shortage disorders.